Free Shipping on Orders over $99 (Excludes )
Kidney Care References
[1] Pizzorno J, et al. The kidney dysfunction epidemic, part 1: Causes. Integr Med (Encinitas). 2015; 14(6): 8-13.
[2] Dusso AS, et al. Vitamin D. Am J Physiol Renal Physiol. 2005; 289(1).
[3] Bhatia D, et al. Mitochondrial dysfunction in kidney injury, inflammation, and disease: potential therapeutic approaches. Kidney Res Clin Pract. 2020; 39(3): 244-258.
[4] Cockwell P and Fisher LA. The global burden of chronic kidney disease. The Lancet. 2020; 395(10225): P662-664.
[5] Genchi G, et al. The effects of cadmium toxicity. Int J Environ Res Public Health. 2020; 17(11): 3782.
[6] Harari F, et al. Blood lead levels and decreased kidney function in a population-based cohort. Am J Kidney Dis. 2018; 72(3): P381-389.
[7] Li SJ, et al. Mercury-induced membranous nephropathy: clinical and pathological features. Clin J Am Soc Nephrol. 2010; 5(3): 439-444.
[8] Vicente-Vicente L, et al. Nephrotoxicity of uranium: Pathophysiological, diagnostic and therapeutic perspectives. Toxicol Sci. 2010; 118(2): 324-347.
[9] Wang YL, et al. The kidney-related effects of polystyrene microplastics on human kidney proximal tubular epithelial cells HK-2 and male C57BL/6 mice. Environ Health Perspect. 2021; 129(5): https://doi.org/10.1289/EHP7612.
[10] Virzi GM, et al. Endotoxin effects on cardiac and renal functions and cardiorenal syndromes. Blood Purif. 2017; 44: 314-326.
[11] Pecoits-Filho R, et al. Interactions between kidney disease and diabetes: dangerous liaisons. Diabetol Metab Syndr. 2016; 8(50): https://doi.org/10.1186/s13098-016-0159-z.
[12] Stavropoulou E, et al. Focus on the gut–kidney axis in health and disease. Front Med Nephrol. 2021. https://doi.org/10.3389/fmed.2020.620102.
[13] Gunatilake S, et al. Glyphosate’s synergistic toxicity in combination with other factors as a cause of chronic kidney disease of unknown origin. Int J Environ Res Public Health. 2019; 16(15): 2734.
[14] Clare BA, Conroy RS, Spelman K. The Diuretic effect in human subjects of an extract of Taraxacum officinale Folium over a single day. J Altern Complement Med. 2009; 15(8): 929-934.
[15] Sharmila R et al. Nephroprotective effect of β-sitosterol on N-diethylnitrosamine initiated and ferric nitrilotriacetate promoted acute nephrotoxicity in Wistar rats. J Basic Clin Physiol Pharmacol. 2016; 27(5): https://doi.org/10.1515/jbcpp-2015-0085.
[16] Sharmila R and Sindhu G. Modulation of angiogenesis, proliferative response and apoptosis by β-sitosterol in rat model of renal carcinogenesis. Indian J Clin Biochem. 2017; 32(2): 142-152.
[17] Munoz-Reyes D et al. Transit and metabolic pathways of quercetin in tubular cells: Involvement of its antioxidant properties in the kidney. Antioxidants (Basel). 2021; 10(6): 909.
[18] El-Twab SMA. Chicoric acid prevents methotrexate-induced kidney injury by suppressing NF-κB/NLRP3 inflammasome activation and up-regulating Nrf2/ARE/HO-1 signaling. Inflamm Res. 2019; 68(6): 511-523.
[19] Fursenco C, et al. Solidago virgaurea L.: A review of its ethnomedicinal uses, phytochemistry, and pharmacological activities. Biomolecules. 2020; 10(12): 1619.
[20] Chen DQ, et al. Combined melatonin and poricoic acid A inhibits renal fibrosis through modulating the interaction of Smad3 and β-catenin pathway in AKI-to-CKD continuum. Ther Adv Chronic Dis. 2019; 10: 2040622319869116.
[21] Wu ZL, et al. Sclederma of Poria cocos exerts its diuretic effect via suppression of renal aquaporin-2 expression in rats with chronic heart failure. J Ethnopharmacol. 2014; 155(1): 563-571.
[22] Zhao YY, et al. Urinary metabonomic study of the surface layer of Poria cocos as an effective treatment for chronic renal injury in rats. J Ethnopharmacol. 2013; 148(2): 403-410.
[23] Zhao YY. Traditional uses, phytochemistry, pharmacology, pharmacokinetics, and quality control of Polyporus umbellatus (Pers.) Fries: a review. J Ethnopharmacol. 2013; 149(1): 35-48.
[24] Zhang W, et al. Diuretic activity and kidney medulla AQP1, AQP2, AQP3, V2R expression of the aqueous extract of sclerotia of Polyporus umbellatus FRIES in normal rats. J Ethnopharmacol. 2010; 128(2): 433-437.
[25] Wang YN, et al. Polyporus umbellatus protects against renal fibrosis by regulating intrarenal fatty acyl metabolites. Front Pharmacol. 2021. 12: 633566.
[26] Lin EY, et al. The natural compound 2,3,5,4′-tetrahydroxystilbene-2-O-β-d glucoside protects against adriamycin-induced nephropathy through activating the Nrf2-Keap1 antioxidant pathway. Environ Toxicol. 2018; 33(1): 72-82. doi: 10.1002/tox.22496.
[27] Qi W, et al. Astragaloside IV attenuates glycated albumin-induced epithelial-to-mesenchymal transition by inhibiting oxidative stress in renal proximal tubular cells. Cell Stress Chaperones. 2014; 19(1): 105-114.
[28] Ju Y, et al. Protective effects of Astragaloside IV on endoplasmic reticulum stress-induced renal tubular epithelial cells apoptosis in type 2 diabetic nephropathy rats. Biomed Pharmacother. 2019; 109: 84-92.
[29] Guo H, et al. Astragaloside IV protects against podocyte injury via SERCA2-dependent ER stress reduction and AMPKα-regulated autophagy induction in streptozotocin-induced diabetic nephropathy. Sci Rep. 2017; 7(1): 6852.
[30] Xing L, et al. Astragaloside IV protects against podocyte apoptosis by inhibiting oxidative stress via activating the PPARγ-Klotho-FoxO1 axis in diabetic nephropathy. Life Sci. 2021; 269(15): 119068.
[31] Buchanan S, et al. Klotho, aging, and the failing kidney. Front Endocrinol. 2020.
[32] Kuro-O M, et al. Phosphate and Klotho. Kidney Int Suppl. 2011; 79(S121): S20-S23.
[33] Wang X, et al. Astragaloside IV improves renal function and fibrosis via inhibition of miR-21-induced podocyte dedifferentiation and mesangial cell activation in diabetic mice. Drug Des Devel Ther. 2018; 12: 2431-2442.
[34] Chowdhury S, et al. Ferulic acid protects hyperglycemia-induced kidney damage by regulating oxidative insult, inflammation, and autophagy. Front Pharmacol. 2019; 10: 27.
[35] Zhou Q, et al. Ferulic Acid Protected from Kidney Ischemia-Reperfusion Injury in Mice: Possible Mechanism Through Increasing Adenosine Generation via HIF-1α. Inflammation. 2018; 41(6): 2068-2078.